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Its production can be regulated by various factors like C/EBP (Hungness et al., 2002) and PPAR-activated proteins ANGPTL4 (Phua et al., 2017), CIDEA (Chatterjee et al., 2015), and FATP1 (Nishiyama et al., 2018)
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Consistently, our recent genome-wide CRISPR screens performed in the presence of mitochondrial inhibitors also identified that cell fitness defect in the SLC25A19 KOs can be buffered by a spectrum of mitochondrial inhibitors (Supplementary Fig
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